|
1）Yan WX, Archelos JJ, Hartung H-P, et al. P0 protein is a target antigen in chronic inflammatory demyelinating polyradiculoneuropathy. Ann Neurol. 2001; 50: 286-92
|
|
|
|
2）Gabriel CM, Gregson NA, Hughes RA. Anti-PMP22 antibodies in patients with inflammatory neuropathy. J Neuroimmunol. 2000; 104: 139-46
|
|
|
|
|
|
3）Connolly AM, Pestronk A, Trotter JL, et al. High-titer selective serum anti-beta-tubulin antibodies in chronic inflammatory demyelinating polyneuropathy. Neurology. 1993; 43: 557-62
|
|
|
|
|
|
4）Kuwahara M, Suzuki H, Samukawa M, et al. Clinical features of CIDP with LM1-associated antibodies. J Neurol Neurosurg Psychiatry. 2013; 84: 573-5
|
|
|
|
|
|
5）Fredman P, Vedeler CA, Nyland H, et al. Antibodies in sera from patients with inflammatory demyelinating polyradiculoneuropathy react with ganglioside LM1 and sulphatide of peripheral nerve myelin. J Neurol. 1991; 238: 75-9
|
|
|
|
|
|
6）Salzer JL, Brophy PJ, Peles E. Molecular domains of myelinated axons in the peripheral nervous system. Glia. 2008; 56: 1532-40
|
|
|
|
|
|
7）Buttermore ED, Thaxton CL, Bhat MA. Organization and maintenance of molecular domains in myelinated axons. J Neurosci. Res. 2013; 91: 603-22
|
|
|
|
|
|
8）Feinberg K, Eshed-Eisenbach Y, Frechter S, et al. A glial signal consisting of gliomedin and NrCAM clusters axonal Na+ channels during the formation of nodes of Ranvier. Neuron. 2010; 65: 490-502
|
|
|
|
|
|
9）Thaxton C, Pillai AM, Pribisko AL, et al. Nodes of Ranvier act as barriers to restrict invasion of flanking paranodal domains in myelinated axons. Neuron. 2011; 69: 244-57
|
|
|
|
|
|
10）Sherman DL, Tait S, Melrose S, et al. Neurofascins are required to establish axonal domains for saltatory conduction. Neuron. 2005; 48: 737-42
|
|
|
|
|
|
11）Boyle MET, Berglund EO, Murai KK, et al. Contactin orchestrates assembly of the septate-like junctions at the paranode in myelinated peripheral nerve. Neuron. 2001; 30: 385-97
|
|
|
|
|
|
12）Pillai AM, Thaxton C, Pribisko AL, et al. Spatiotemporal ablation of myelinating glia-specific neurofascin (Nfasc NF155)in mice reveals gradual loss of paranodal axoglial junctions and concomitant disorganization of axonal domains. J Neurosci Res. 2009; 87: 1773-93
|
|
|
|
|
|
13）Traka M, Goutebroze L, Denisenko N, et al. Association of TAG-1 with Caspr2 is essential for the molecular organization of juxtaparanodal regions of myelinated fibers. J Cell Biol. 2003; 162: 1161-72
|
|
|
|
|
|
14）Lonigro A, Devaux JJ. Disruption of neurofascin and gliomedin at nodes of Ranvier precedes demyelination in experimental allergic neuritis. Brain. 2009; 132: 260-73
|
|
|
|
|
|
15）Devaux JJ, Odaka M, Yuki N. Nodal proteins are target antigens in Guillain-Barré syndrome. J Peripher Nerv Syst. 2012; 17: 62-71
|
|
|
|
|
|
16）Devaux JJ. Antibodies to gliomedin cause peripheral demyelinating neuropathy and the dismantling of the nodes of Ranvier. Am J Pathol. 2012; 181: 1402-13
|
|
|
|
|
|
17）Ng JKM, Malotka J, Kawakami N, et al. Neurofascin as a target for autoantibodies in peripheral neuropathies. Neurology. 2012; 79: 2241-8
|
|
|
|
|
|
18）Querol L, Nogales-Gadea G, Rojas-Garcia R, et al. Neurofascin IgG4 antibodies in CIDP associate with disabling tremor and poor response to IVIg. Neurology. 2014; 82: 879-86
|
|
|
|
|
|
19）Ogata H, Yamasaki R, Hiwatashi A, et al. Characterization of IgG4 anti-neurofascin 155 antibody-positive polyneuropathy. Ann Clin Transl Neurol. 2015; 2: 960-71
|
|
|
|
|
|
20）Notturno F, Di Febo T, Yuki N, et al. Autoantibodies to neurofascin-186 and gliomedin in multi-focal motor neuropathy. J Neuroimmunol. 2014; 276: 207-12
|
|
|
|
|
|
21）Doppler K, Appeltshauser L, Krämer HH, et al. Contactin-1 and Neurofascin-155/-186 Are Not Targets of Auto-Antibodies in Multifocal Motor Neuropathy. PLoS One. 2015; 10: e0134274
|
|
|
|
|
|
22）Querol L, Nogales-Gadea G, Rojas-Garcia R, et al. Antibodies to contactin-1 in chronic inflammatory demyelinating polyneuropathy. Ann. Neurol. 2013; 73: 370-80
|
|
|
|
|
|
23）Doppler K, Appeltshauser L, Wilhelmi K, et al. Destruction of paranodal architecture in inflammatory neuropathy with anti-contactin-1 autoanti-bodies. J Neurol Neurosurg Psychiatry. 2015; 1: 1-9
|
|
|
|
|
|
24）Miura Y, Devaux JJ, Fukami Y, et al. Contactin 1 IgG4 associates to chronic inflammatory demyelinating polyneuropathy with sensory ataxia. Brain. 2015; 138: 1484-91
|
|
|
|
|
|
25）Labasque M, Hivert B, Nogales-Gadea G, et al. Specific contactin N-glycans are implicated in neurofascin binding and autoimmune targeting in peripheral neuropathies. J Biol Chem. 2014; 289: 7907-18
|
|
|
|
|
|
26）Prüss H, Schwab JM, Derst C, et al. Neurofascin as target of autoantibodies in Guillain-Barre syndrome. Brain. 2011; 134: e173; author reply e174
|
|
|
|
|
|
27）Yan W, Nguyen T, Yuki N, et al. Antibodies to neurofascin exacerbate adoptive transfer experimental autoimmune neuritis. J Neuroimmunol. 2014; 277: 13-7
|
|
|
|
|
|
28）Kawamura N, Yamasaki R, Yonekawa T, et al. Anti-neurofascin antibody in patients with combined central and peripheral demyelination. Neurology. 2013; 81: 714-22
|
|
|
|
|
|
29）Huijbers MG, Querol LA, Niks EH, et al. The expanding field of IgG4-mediated neurological autoimmune disorders. Eur J Neurol. 2015; 22: 1151-61
|
|
|
|
|
|
30）Kwa MSG, Van Schaik IN, Brand A, et al. Investigation of serum response to PMP22, connexin 32 and P0 in inflammatory neuropathies. J Neuroimmunol. 2001; 116: 220-5
|
|
|
|
|
|
31）Derfuss T, Parikh K, Velhin S, et al. Contactin-2/TAG-1-directed autoimmunity is identified in multiple sclerosis patients and mediates gray matter pathology in animals. Proc Natl Acad Sci U S A. 2009; 106: 8302-7
|
|
|
|
|
|
32）Lancaster E, Huijbers MGM, Bar V, et al. Investigations of caspr2, an autoantigen of encephalitis and neuromyotonia. Ann Neurol. 2011; 69: 303-11
|
|
|
|
|
|
33）Iijima M, Tomita M, Morozumi S, et al. Single nucleotide polymorphism of TAG-1 influences IVIg responsiveness of Japanese patients with CIDP. Neurology. 2009; 73: 1348-52
|
|
|
|
|