1)Greaves M. Cancer -The Revolutionary Legacy. Oxford University Press.(日本語訳: がん―進化の遺産,水谷修紀監訳,ブレーン出版)
|
|
|
2)Ford AM, Ridge SA, Cabrera ME, et al. In utero rearrangements in the trithorax-related oncogene in infant leukaemias. Nature. 1993; 363: 358-60
|
|
|
3)Ford AM, Bennett CA, Price CM, et al. Fetal origins of the TEL-AML1 fusion gene in identical twins with leukemia. Proc Natl Acad Sci U S A. 1998; 95: 4584-8
|
|
|
4)Mori H, Colman SM, Xiao Z, et al. Chromosome translocations and covert leukemic clones are generated during normal fetal development. Proc Natl Acad Sci U S A. 2002; 99: 8242-7
|
|
|
5)Urayama KY, Ma X, Selvin S, et al. Early life exposure to infections and risk of childhood acute lymphoblastic leukemia. Int J Cancer. 2011; 128: 1632-43
|
|
|
6)Jackson KJ, Kidd MJ, Wang Y, et al. The shape of the lymphocyte receptor repertoire: lessons from the B cell receptor. Front Immunol. 2013; 4: 263
|
|
|
7)Papaemmanuil E, Rapado I, Li Y, et al. RAG-mediated recombination is the predominant driver of oncogenic rearrangement in ETV6-RUNX1 acute lymphoblastic leukemia. Nat Genet. 2014; 46: 116-25
|
|
|
8)Swaminathan S, Klemm L, Park E, et al. Mechanisms of clonal evolution in childhood acute lymphoblastic leukemia. Nat Immunol. 2015; 16: 766-74
|
|
|
9)Mayilaadumveettil N, Sathees CR. Role of recombination activating genes in the generation of antigen receptor diversity and beyond. Immunology. 2012; 137: 271-81
|
|
|