|
1）Harrap SB, Van der Merwe WM, Griffin SA, et al. Brief angiotensin converting enzyme inhibitor treatment in young spontaneously hypertensive rats reduces blood pressure long-term. Hypertension. 1990; 16: 603-14
|
|
|
|
2）Harrap SB. A developmental genetic mechanism involving angiotensin in spontaneously hypertensive rats. Clin Exp Pharmacol Physiol Suppl. 1992; 19: 19-22
|
|
|
|
|
|
3）Wu JN, Berecek KH. Prevention of genetic hypertension by early treatment of spontaneously hypertensive rats with the angiotensin converting enzyme inhibitor captopril. Hypertension. 1993; 22: 139-46
|
|
|
|
|
|
4）Christensen KL, Jespersen LT, Mulvany MJ. Development of blood pressure in spontaneously hypertensive rats after withdrawal of long-term treatment related to vascular structure. J Hypertens. 1989; 7: 83-90
|
|
|
|
|
|
5）Gillies LK, Lu M, Wang H, et al. AT1 receptor antagonist treatment caused persistent arterial functional changes in young spontaneously hypertensive rats. Hypertension. 1997; 30: 1471-8
|
|
|
|
|
|
6）Giudicelli JF, Freslon JL, Glasson S, et al. Captopril and hypertension development in the SHR. Clin Exp Hypertens. 1980; 2: 1083-96
|
|
|
|
|
|
7）Morton JJ, Beattie EC, MacPherson F. Angiotensin II receptor antagonist losartan has persistent effects on blood pressure in the young spontaneously hypertensive rat: lack of relation to vascular structure. J Vasc Res. 1992; 29: 264-9
|
|
|
|
|
|
8）Nakaya H, Sasamura H, Hayashi M, et al. Temporary treatment of prepubescent rats with angiotensin inhibitors suppresses the development of hypertensive nephrosclerosis. J Am Soc Nephrol. 2001; 12: 659-66
|
|
|
|
|
|
9）Peng F, Lin J, Lin L, et al. Transient prehypertensive treatment in spontaneously hypertensive rats: a comparison of losartan and amlodipine regarding long-term blood pressure, cardiac and renal protection. Int J Mol Med. 2012; 30: 1376-86
|
|
|
|
|
|
10）Heijnen BF, Peutz-Kootstra CJ, Mullins JJ, et al. Transient renin-angiotensin system stimulation in an early stage of life causes sustained hypertension in rats. J Hypertens. 2011; 29: 2369-80
|
|
|
|
|
|
11）Heijnen BF, Nelissen J, van Essen H, et al. Irreversible renal damage after transient renin-angiotensin system stimulation: involvement of an AT1-receptor mediated immune response. PLoS One. 2013; 8: e57815
|
|
|
|
|
|
12）Koeners MP, Braam B, Joles JA. Perinatal inhibition of NF-kappaB has long-term antihypertensive effects in spontaneously hypertensive rats. J Hypertens. 2011; 29: 1160-6
|
|
|
|
|
|
13）Harwani SC, Chapleau MW, Legge KL, et al. Neurohormonal modulation of the innate immune system is proinflammatory in the prehypertensive spontaneously hypertensive rat, a genetic model of essential hypertension. Circ Res. 2012; 111: 1190-7
|
|
|
|
|
|
14）Ng K, Butlin M, Avolio AP. Persistent effect of early, brief angiotensin-converting enzyme inhibition on segmental pressure dependency of aortic stiffness in spontaneously hypertensive rats. J Hypertens. 2012; 30: 1782-90
|
|
|
|
|
|
15）Hale TM, Robertson SJ, Burns KD, et al. Short-term ACE inhibition confers long-term protection against target organ damage. Hypertens Res. 2012; 35: 604-10
|
|
|
|
|
16）Biwer LA, Broderick TL, Xu H, et al. Protection against L-NAME-induced reduction in cardiac output persists even after cessation of angiotensin-converting enzyme inhibitor treatment. Acta Physiol (Oxf). 2013; 207: 156-65
|
|
|
|
|
|
17）Dejima T, Tamura K, Wakui H, et al. Prepubertal angiotensin blockade exerts long-term therapeutic effect through sustained ATRAP activation in salt-sensitive hypertensive rats. J Hypertens. 2011; 29: 1919-29
|
|
|
|
|
|
18）Kawarazaki H, Ando K, Nagae A, et al. Mineralocorticoid receptor activation contributes to salt-induced hypertension and renal injury in prepubertal Dahl salt-sensitive rats. Nephrol Dial Transplant. 2010; 25: 2879-89
|
|
|
|
|
|
19）Julius S, Nesbitt SD, Egan BM, et al. Feasibility of treating prehypertension with an angiotensin-receptor blocker. N Engl J Med. 2006; 354: 1685-97
|
|
|
|
|
|
20）Skov K, Eiskjaer H, Hansen HE, et al. Treatment of young subjects at high familial risk of future hypertension with an angiotensin-receptor blocker. Hypertension. 2007; 50: 89-95
|
|
|
|
|
|
21）Smallegange C, Hale TM, Bushfield TL, et al. Persistent lowering of pressure by transplanting kidneys from adult spontaneously hypertensive rats treated with brief antihypertensive therapy. Hypertension. 2004; 44: 89-94
|
|
|
|
|
|
22）Ishiguro K, Hayashi K, Sasamura H, et al. “Pulse” treatment with high-dose angiotensin blocker reverses renal arteriolar hypertrophy and regresses hypertension. Hypertension. 2009; 53: 83-9
|
|
|
|
|
|
23）Sasamura H, Nakaya H, Julius S, et al. Feasibility of regression of hypertension with contemporary antihypertensive agents. Am J Hypertens. 2013; 26: 1381-8
|
|
|
|
|