|
1）Baud L, Ardaillou R. Involvement of reactive oxygen species in kidney damage. Br Med Bull. 1993; 49: 621-9
|
|
|
|
2）Weinberg JM. The cell biology of ischemic renal injury. Kidney Int. 1991; 39: 476-500
|
|
|
|
|
3）Jang HR, Ko GJ, Wasowska BA, et al. The interaction between ischemia-reperfusion and immune responses in the kidney. J Mol Med (Berl). 2009; 87: 859-64
|
|
|
|
|
|
4）Jang HR, Rabb H. The innate immune response in ischemic acute kidney injury. Clin Immunol. 2009; 130: 41-50
|
|
|
|
|
|
5）Arany I, Safirstein RL. Cisplatin nephrotoxicity. Semin Nephrol. 2003; 23: 460-4
|
|
|
|
|
|
6）Townsend DM, Deng M, Zhang L, et al. Metabolism of Cisplatin to a nephrotoxin in proximal tubule cells. J Am Soc Nephrol. 2003; 14: 1-10
|
|
|
|
|
|
7）Santos NA, Bezerra CS, Martins NM, et al. Hydroxyl radical scavenger ameliorates cisplatin-induced nephrotoxicity by preventing oxidative stress, redox state unbalance, impairment of energetic metabolism and apoptosis in rat kidney mitochondria. Cancer Chemother Pharmacol. 2008; 61: 145-55
|
|
|
|
|
|
8）Santos NA, Catao CS, Martins NM, et al. Cisplatin-induced nephrotoxicity is associated with oxidative stress, redox state unbalance, impairment of energetic metabolism and apoptosis in rat kidney mitochondria. Arch Toxicol. 2007; 81: 495-504
|
|
|
|
|
|
9）Brouwers EE, Huitema AD, Schellens JH, et al. The effects of sulfur-containing compounds and gemcitabine on the binding of cisplatin to plasma proteins and DNA determined by inductively coupled plasma mass spectrometry and high performance liquid chromatography-inductively coupled plasma mass spectrometry. Anticancer Drugs. 2008; 19: 621-30
|
|
|
|
|
|
10）Chang G, Chen L, Lin HM, et al. Nox4 inhibition enhances the cytotoxicity of cisplatin in human renal cancer cells. J Exp Ther Oncol. 2012; 10: 9-18
|
|
|
|
|
|
11）Seeliger E, Sendeski M, Rihal CS, et al. Contrast-induced kidney injury: mechanisms, risk factors, and prevention. Eur Heart J. 2012; 33: 2007-15
|
|
|
|
|
|
12）Wong PC, Li Z, Guo J, et al. Pathophysiology of contrast-induced nephropathy. Int J Cardiol. 2012; 158: 186-92
|
|
|
|
|
|
13）Kobori H, Nangaku M, Navar LG, et al. The intrarenal renin-angiotensin system: from physiology to the pathobiology of hypertension and kidney disease. Pharmacol Rev. 2007; 59: 251-87
|
|
|
|
|
|
14）Jennings BL, Anderson LJ, Estes AM, et al. Involvement of cytochrome P-450 1B1 in renal dysfunction, injury, and inflammation associated with angiotensin II-induced hypertension in rats. Am J Physiol Renal Physiol. 2012; 302: F408-20
|
|
|
|
|
|
15）Pallone TL, Zhang Z, Rhinehart K. Physiology of the renal medullary microcirculation. Am J Physiol Renal Physiol. 2003; 284: F253-66
|
|
|
|
|
|
16）Mori T, Ogawa S, Cowely AW, Jr., et al. Role of renal medullary oxidative and/or carbonyl stress in salt-sensitive hypertension and diabetes. Clin Exp Pharmacol Physiol. 2012; 39: 125-31
|
|
|
|
|
|
17）Zhang A, Jia Z, Guo X, et al. Aldosterone induces epithelial-mesenchymal transition via ROS of mitochondrial origin. Am J Physiol Renal Physiol. 2007; 293: F723-31
|
|
|
|
|
|
18）Fan YY, Kohno M, Hitomi H, et al. Aldosterone/mineralocorticoid receptor stimulation induces cellular senescence in the kidney. Endocrinology. 2011; 152: 680-8
|
|
|
|
|
|
19）Duranton F, Cohen G, De SR, et al. Normal and pathologic concentrations of uremic toxins. J Am Soc Nephrol. 2012; 23: 1258-70
|
|
|
|
|
|
20）Chiang CK, Tanaka T, Nangaku M. Dysregulated oxygen metabolism of the kidney by uremic toxins: review. J Ren Nutr. 2012; 22: 77-80
|
|
|
|
|
|
21）Bolati D, Shimizu H, Yisireyili M, et al. Indoxyl sulfate, a uremic toxin, downregulates renal expression of Nrf2 through activation of NF-kappaB. BMC Nephrol. 2013; 14: 56
|
|
|
|
|
|
22）Lassegue B, San MA, Griendling KK. Biochemistry, physiology, and pathophysiology of NADPH oxidases in the cardiovascular system. Circ Res. 2012; 110: 1364-90
|
|
|
|
|
|
23）Niwa T, Shimizu H. Indoxyl sulfate induces nephrovascular senescence. J Ren Nutr. 2012; 22: 102-6
|
|
|
|
|
|
24）Watanabe H, Miyamoto Y, Honda D, et al. p-Cresyl sulfate causes renal tubular cell damage by inducing oxidative stress by activation of NADPH oxidase. Kidney Int. 2013; 83: 582-92
|
|
|
|
|
|
25）Gorin Y. Nox4 as a potential therapeutic target for treatment of uremic toxicity associated to chronic kidney disease. Kidney Int. 2013; 83: 541-3
|
|
|
|
|
|
26）Hashimoto T, Perlot T, Rehman A, et al. ACE2 links amino acid malnutrition to microbial ecology and intestinal inflammation. Nature. 2012; 487: 477-81
|
|
|
|
|