1)Kearney PM, Whelton M, Reynolds K, et al. Global burden of hypertension: analysis of worldwide data. Lancet. 2005; 365: 217-23
|
|
|
2)Kupper N, Willemsen G, Riese H, et al. Heri-tability of daytime ambulatory blood pressure in an extended twin design. Hypertension. 2005; 45: 80-5
|
|
|
3)Takeuchi F, Yamamoto K, Katsuya T, et al. Reevaluation of the association of seven candi-date genes with blood pressure and hypertension: a replication study and meta-analysis with a larger sample size. Hypertens Res. 2012; 35: 825-31
|
|
|
4)Kohara K, Tabara Y, Nakura J, et al. Identifi-cation of hypertension-susceptibility genes and pathways by a systemic multiple candidate gene approach: the millennium genome project for hypertension. Hypertens Res. 2008; 31: 203-12
|
|
|
5)Tabara Y, Kohara K, Kita Y, et al. Common variants in the ATP2B1 gene are associated with susceptibility to hypertension: the Japanese Millennium Genome Project. Hypertension. 2010; 56: 973-80
|
|
|
6)Takeuchi F, Isono M, Katsuya T, et al. Blood pressure and hypertension are associated with 7 loci in the Japanese population. Circulation. 2010; 121: 2302-9
|
|
|
7)Tabara Y, Kohara K, Miki T. Hunting for genes for hypertension: the Millennium Genome Project for Hypertension. Hypertens Res. 2012; 35: 567-73
|
|
|
8)Levy D, Ehret GB, Rice K, et al. Genome-wide association study of blood pressure and hyper-tension. Nat Genet. 2009; 41: 677-87
|
|
|
9)Cho YS, Go MJ, Kim YJ, et al. A large-scale genome-wide association study of Asian popula-tions uncovers genetic factors influencing eight quantitative traits. Nat Genet. 2009; 41: 527-34
|
|
|
10)Kobayashi Y, Hirawa N, Tabara Y, et al. Mice lacking hypertension candidate gene ATP2B1 in vascular smooth muscle cells show significant blood pressure elevation. Hypertension. 2012; 59: 854-60
|
|
|
11)Manolio TA, Collins FS, Cox NJ, et al. Finding the missing heritability of complex diseases. Nature. 2009; 461: 747-53
|
|
|
12)International Consortium for Blood Pressure Genome-Wide Association Studies, Ehret GB, et al. Genetic variants in novel pathways influence blood pressure and cardiovascular disease risk. Nature. 2011; 478: 103-9
|
|
|
13)Kato N, Takeuchi F, Tabara Y, et al. Meta-analysis of genome-wide association studies identifies common variants associated with blood pressure variation in east Asians. Nat Genet. 2011; 43: 531-8
|
|
|
14)Bustamante CD, Burchard EG, De la Vega FM. Genomics for the world. Nature. 2011; 475: 163-5
|
|
|
15)Padmanabhan S, Newton-Cheh C, Dominiczak AF. Genetic basis of blood pressure and hypertension. Trends Genet. 2012; 28: 397-408
|
|
|
16)Simon DB, Nelson-Williams C, Bia MJ, et al. Gitelmans variant of Bartters syndrome, inherited hypokalaemic alkalosis, is caused by mutations in the thiazide-sensitive Na-Cl cotransporter. Nat Genet. 1996; 12: 24-30
|
|
|
17)Simon DB, Karet FE, Hamdan JM, et al. Bartters syndrome, hypokalaemic alkalosis with hyper-calciuria, is caused by mutations in the Na-K-2Cl cotransporter NKCC2. Nat Genet. 1996; 13: 183-8
|
|
|
18)Ji W, Foo JN, ORoak BJ, et al. Rare independent mutations in renal salt handling genes contribute to blood pressure variation. Nat Genet. 2008; 40: 592-9
|
|
|
19)Newton-Cheh C, Johnson T, Gateva V, et al; Wellcome Trust Case Control Consortium, Brown M, Dominiczak A, Newhouse SJ, et al. Genome-wide association study identifies eight loci associated with blood pressure. Nat Genet. 2009; 41: 666-76
|
|
|
20)Johnson T, et al; Cardiogenics Consortium, Fowkes FG, et al; Global BPgen Consortium, Braund PS, et al. Blood pressure loci identified with a gene-centric array. Am J Hum Genet. 2011; 89: 688-700
|
|
|
21)Padmanabhan S, Melander O, Johnson T, et al. Global BPgen Consortium, Munroe P, Caulfield MJ, Zanchetti A, et al. Genome-wide association study of blood pressure extremes identifies variant near UMOD associated with hyperten-sion. PLoS Genet. 2010; 6: e1001177
|
|
|