|
1) Onouchi Y, Tamari M, Takahashi A, et al. A genomewide linkage analysis of Kawasaki disease: evidence for linkage to chromosome 12. J Hum Genet. 2007; 52: 179-90
|
|
|
|
2) Onouchi Y, Gunji T, Burns JC, et al. ITPKC functional polymorphism associated with Kawasaki disease susceptibility and formation of coronary artery aneurysms. Nat Genet. 2008; 40: 35-42
|
|
|
|
|
|
3) Sabbagh L, Bourbonnière M, Sékaly RP, et al. Selective up-regulation of caspase-3 gene expression following TCR engagement. Mol Immunol. 2005; 42: 1345-54
|
|
|
|
|
|
4) Woo M, Hakem R, Soengas MS, et al. Essential contribution of caspase 3/CPP32 to apoptosis and its associated nuclear changes. Genes Dev. 1998; 12: 806-19
|
|
|
|
|
|
5) Woo M, Hakem R, Furlonger C, et al. Caspase-3 regulates cell cycle in B cells: a consequence of substrate specificity. Nat Immunol. 2003; 4: 1016-22
|
|
|
|
|
|
6) Onouchi Y, Ozaki K, Buns JC, et al. Common variants in CASP3 confer susceptibility to Kawasaki disease. Hum Mol Genet. 2010; 19: 2898-906
|
|
|
|
|
|
7) Macián F, García-Cózar F, Im SH, et al. Transcriptional mechanisms underlying lympho-cyte tolerance. Cell. 2002; 109: 719-31
|
|
|
|
|
|
8) Wu W, Misra RS, Russell JQ, et al. Proteolytic regulation of nuclear factor of activated T (NFAT) c2 cells and NFAT activity by caspase-3. J Biol Chem. 2006; 281: 10682-90
|
|
|
|
|
|
9) Burgner D, Davila S, Breunis WB, et al. A genome-wide association study identifies novel and functionally related susceptibility Loci for Kawasaki disease. PLoS Genet. 2009; 5: e1000319
|
|
|
|
|