|
1) Date Y, et al. Peripheral ghrelin transmits orexigenic signals through the noradrenergic pathway from the hindbrain to the hypotha-lamus. Cell Metab. 2006; 4: 323-31
|
|
|
|
2) 清水弘行, 森 昌朋. D. 肥満 Ⅳ. エネルギー代謝とその調節機序 組織間ネットワークによる代謝調節. 新時代の糖尿病学(2) —病因・診断・治療研究の進歩—. 日本臨牀. 2008; 66: 650-5
|
|
|
|
|
3) Oh-I S, et al. Identification of nesfatin-1 as a satiety molecule in the hypothalamus. Nature. 2006; 443: 709-12
|
|
|
|
|
|
4) Brailoiu GC, et al. Nesfatin-1: distribution and interaction with a G protein-coupled receptor in the rat brain. Endocrinology. 2007; 148: 5088-94
|
|
|
|
|
|
5) Kohno D, et al. Nesfatin-1 neurons in para-ventricular and supraoptic nuclei of the rat hypo-thalamic coexpress oxytocin and vasopressin and are activated by refeeding. Endocrinology. 2008; 149: 1295-301
|
|
|
|
|
|
6) Fort P, et al. The satiety molecule nesfatin-1 is co-expressed within melanin concentrating hormone in tuberal hypothalamic neurons of the rat. Neuroscience. 2008; 155: 174-81
|
|
|
|
|
|
7) Pan, W. et al. Nesfatin-1 crosses the blood-brain barrier without saturation. Peptides. 2007; 28: 2223-8
|
|
|
|
|
|
8) Price TO, et al. Permeability of the blood-brain barrier to a novel satiety molecule nesfatin-1. Peptides 2007; 28: 2372-81
|
|
|
|
|
|
9) Shimizu H, et al. Peripheral administration of nesfatin-1 reduces food intake in mice: the leptin-independent mechanism. Endocrinology. 2009; 150: 662-71
|
|
|
|
|
|
10) Shimizu H, et al. A new anorexigenic protein, nesfatin-1. Peptides. 2009; 30: 995-8
|
|
|
|
|
|
11) Stengel A, et al. Identification and charac-terization of nesfatin-1 immunoreactivity in endocrine cell type of the rat gastric oxyntic mucosa. Endocrinology. 2009; 150: 232-8
|
|
|
|
|