1) Ohmuraya M, Hirota M, Araki M, et al. Autophagic cell death of pancreatic acinar cells in serine protease inhibitor Kazal type 3-deficient mice. Gastroenterology. 2005; 129: 696-705
|
|
|
2) Ohmuraya M, Hirota M, Araki K, et al. Enhanced trypsin activity in pancreatic acinar cells deficient for serine protease inhibitor Kazal type 3. Pancreas. 2006; 33: 104-6
|
|
|
3) Hashimoto D, Ohmuraya M, Hirota M, et al. Involvement of autophagy in trypsinogen activation within the pancreatic acinar cells. J Cell Biol. 2008; 181: 1065-72
|
|
|
4) 大村谷昌樹, 広田昌彦, 橋本大輔, 他. 遺伝子改変マウスを用いた膵炎の発症機構の解析. 膵臓. 2008; 23: 20-4
|
|
|
5) 廣田昌彦, 橋本大輔, 大村谷昌樹, 他. 急性膵炎発症の細胞内イベント. 肝胆膵. 2009; 59: 165-71
|
|
|
6) Hirota M, Ohmuraya M, Baba H. Genetic background of pancreatitis. Postgraduate Med J. 2006; 82: 775-8
|
|
|
7) Hirota M, Ohmuraya M, Baba H. The role of trypsin, trypsin inhibitor, and trypsin receptor in the onset of pancreatitis. J Gastroenterol. 2006; 41: 832-6
|
|
|
8) Liddle RA. Pathophysiology of SPINK mutations in pancreatic development and disease. Endocrinol Metab Clin North Am. 2006; 35: 345-56
|
|
|
9) Criddle DN, McLaughlin E, Murphy JA, et al. The pancreas misled: signals to pancreatitis. Pancreatology. 2007; 7: 436-46
|
|
|