|
1) Li K, Foy E, Ferreon JC, et al. Immune evasion by hepatitis C virus NS3/4A protease-mediated cleavage of the Toll-like receptor 3 adaptor protein TRIF. Proc Natl Acad Sci U S A. 2005; 102: 2992-7
|
|
|
|
2) Ferreon JC, Ferreon AC, Li K, et al. Molecular determinants of TRIF proteolysis mediated by the hepatitis C virus NS3/4A protease. J Biol Chem. 2005; 280: 20483-92
|
|
|
|
|
|
3) Foy E, Li K, Sumpter R Jr, et al. Control of antiviral defenses through hepatitis C virus disruption of retinoic acid-inducible gene-I signaling. Proc Natl Acad Sci U S A. 2005; 102: 2986-91
|
|
|
|
|
|
4) Loo YM, Owen DM, Li K, et al. Viral and therapeutic control of IFN-beta promoter stimulator 1 during hepatitis C virus infection. Proc Natl Acad Sci U S A. 2006; 103: 6001-6
|
|
|
|
|
|
5) Tasaka M, Sakamoto N, Itakura Y, et al. Hepatitis C virus non-structural proteins responsible for suppression of the RIG-I/Cardif-induced interferon response. J Gen Virol. 2007; 88: 3323-33
|
|
|
|
|
|
6) Asahina Y, Izumi N, Hirayama I, et al. Potential relevance of cytoplasmic viral sensors and related regulators involving innate immunity in antiviral response. Gastroenterology. 2008; 134: 1396-405
|
|
|
|
|
|
7) Saito T, Owen DM, Jiang F, et al. Innate immunity induced by composition-dependent RIG-I recognition of hepatitis C virus RNA. Nature. 2008; 454: 523-7
|
|
|
|
|
|
8) Blindenbacher A, Duong FH, Hunziker L, et al. Expression of hepatitis C virus proteins inhibits interferon alpha signaling in the liver of transgenic mice. Gastroenterology. 2003; 124: 14565-75
|
|
|
|
|
|
9) Hosui A, Ohkawa K, Ishida H, et al. Hepatitis C virus core protein differently regulates the JAK-STAT signaling pathway under interleukin-6 and interferon-gamma stimuli. J Biol Chem. 2003; 278: 28562-71
|
|
|
|
|
|
10) Lin W, Kim SS, Yeung E, et al. Hepatitis C virus core protein blocks interferon signaling by interaction with the STAT1 SH2 domain. J Virol. 2006; 80: 9226-35
|
|
|
|
|
|
11) Lin W, Choe WH, Hiasa Y, et al. Hepatitis C virus expression suppresses interferon signaling by degrading STAT1. Gastroenterology. 2005; 128: 1034-41
|
|
|
|
|
|
12) Akuta N, Suzuki F, Kawamura Y, et al. Amino acid substitutions in the hepatitis C virus core region are the important predictor of hepatocarcinogenesis. Hepatology. 2007; 46: 1357-64
|
|
|
|
|
|
13) Takehara T, Hayashi N. Natural killer cells in hepatitis C virus infection: from innate immunity to adaptive immunity. Clin Gastroenterol Hepatol. 2005; 3: S78-81
|
|
|
|
|
|
14) Jinushi M, Takehara T, Tatsumi T, et al. Negative regulation of NK cell activities by inhibitory receptor CD94/NKG2A leads to altered NK cell-induced modulation of dendritic cell functions in chronic hepatitis C virus infection. J Immunol. 2004; 173: 6072-81
|
|
|
|
|
|
15) Nattermann J, Feldmann G, Ahlenstiel G, et al. Surface expression and cytolytic function of natural killer cell receptors is altered in chronic hepatitis C. Gut. 2006; 55: 869-77
|
|
|
|
|
|
16) Golden-Mason L, Madrigal-Estebas L, McGrath E, et al. Altered natural killer cell subset distributions in resolved and persistent hepatitis C virus infection following single source exposure. Gut. 2008; 57: 1121-8
|
|
|
|
|
|
17) Golden-Mason L, Castelblanco N, O'Farrelly C, et al. Phenotypic and functional changes of cytotoxic CD56pos natural T cells determine outcome of acute hepatitis C virus infection. J Virol. 2007; 81: 9292-8
|
|
|
|
|
|
18) Nattermann J, Sherzada R, Iwan A, et al. Hepatitis C virus-induced secretion of inflammatory chemokines preferentially recruits NKG2A＋CD8＋ T cells. J Infect Dis. 2008; 198: 213-7
|
|
|
|
|
|
19) Golden-Mason L, Klarquist J, Wahed AS, et al. Cutting edge: programmed death-1 expression is increased on immunocytes in chronic hepatitis C virus and predicts failure of response to antiviral therapy: race-dependent differences. J Immunol. 2008; 180: 3637-41
|
|
|
|
|
|
20) Jinushi M, Takehara T, Tatsumi T, et al. Expression and role of MICA and MICB in human hepatocellular carcinomas and their regulation by retinoic acid. Int J Cancer. 2003; 104: 354-61
|
|
|
|
|
|
21) Jinushi M, Takehara T, Tatsumi T, et al. Impairment of natural killer cell and dendritic cell functions by the soluble form of MHC class I-related chain A in advanced human hepatocellular carcinomas. J Hepatol. 2005; 43: 1013-20
|
|
|
|
|
|
22) Kohga K, Takehara T, Tatsumi T, et al. Serum levels of soluble MHC class I-related chain A in patients with chronic liver diseases and the changes during transcatheter arterial embolization for hepatocellular carcinoma. Cancer Sci. 2008; 99: 1643-9
|
|
|
|
|
|
23) Kaiser BK, Yim D, Chow IT, et al. Disulphide-isomerase-enabled shedding of tumour-associated NKG2D ligands. Nature. 2007; 447: 482-6
|
|
|
|
|
|
24) Khakoo SI, Thio CL, Martin MP, et al. HLA and NK cell inhibitory receptor genes in resolving hepatitis C virus infection. Science. 2004; 305: 872-4
|
|
|
|
|
|
25) Romero V, Azocar J, Zuniga J, et al. Interaction of NK inhibitory receptor genes with HLA-C and MHC class II alleles in Hepatitis C virus infection outcome. Mol Immunol. 2008; 45: 2429-36
|
|
|
|
|
|
26) Li Y, Zhang T, Ho C, et al. Natural killer cells inhibit hepatitis C virus expression. J Leukoc Biol. 2004; 76: 1171-9
|
|
|
|
|
|
27) Wang SH, Huang CX, Ye L, et al. Natural killer cells suppress full cycle HCV infection of human hepatocytes. J Viral Hepat (in press)
|
|
|
|
|
|
28) Jinushi M, Takehara T, Tatsumi T, et al. Natural killer cell and hepatic cell interaction via NKG2A leads to dendritic cell-mediated induction of CD4＋ CD25＋ T cells with PD-1-dependent regulatory activities. Immunology. 2007; 120: 73-82
|
|
|
|
|