1) 川崎病研究グループ(中村好一, 柳川 洋, 川崎富作). 第18回川崎病全国調査成績. 2005
|
|
|
2) De Bruyne B, Bartunek J, Sys SU, et al. Relation between myocardial fractional flow reserve calculated from coronary pressure measurements and exercise-induced myocardial ischemia. Circulation. 1995; 92: 39-46
|
|
|
3) Ogawa S, Ohkubo T, Fukazawa R, et al. Estimation of myocardial hemodynamics before and after intervention in children with Kawasaki disease. J Am Coll Cardiol. 2004; 43: 653-61
|
|
|
4) Segal J, Kern MJ, Scott NA, et al. Alterations of phasic coronary flow velocity in human during percutaneous coronary angioplasty. J Am Coll Cardiol. 1992; 20: 276-86
|
|
|
5) Ofili EO, Kern MJ, Labovitz AJ, et al. Analysis of coronary blood flow velocity dynamics in angio-graphycally normal and stenosed arteries before and after endolumen enlargement by angioplasty. J Am Coll Cardiol. 1993; 21: 308-16
|
|
|
6) Donohoue TJ, Kern MJ, Aguirre FV, et al. Assessing the hemodynamic significance of coronary artery stenoses: analysis of translesional pressure-flow velocity relations in patients. J Am Coll Cardiol. 1993; 22: 449-58
|
|
|
7) Ohkubo T, Fukazawa R, Ikegami E, et al. Reduced shear stress and disturbed flow may lead to coronary aneurysm and thrombus formations. Pediatrics International. 2007; 49: 1-7
|
|
|
8) Pijls NHJ, van Son JAM, Kirkeeide RL, et al. Experimental basis of determining maximum coronary, myocardial, and collateral blood flow by pressure measurement for assessing functional stenosis severity before and after percutaneous transluminal coronary angioplasty. Circulation. 1993; 87: 1354-67
|
|
|
9) Takagi A, Tsurumi Y, Ishii Y, et al. Clinical potential of intravascular ultrasound for physio-logical assessment of coronary stenosis. Circulation. 1999; 100: 250-5
|
|
|
10) Ogawa S, Fukazawa R, Kamisago M, et al. Long-term reduced coronary micro-circulation improves after coronary intervention in children with Kawasaki disease. Eur Heart J. 2007; 28 Suppl: 671
|
|
|
11) Alshihabi S, Chang Y, Fragos J, et al. Shear stress-induced release of PGE2 and PGI2 by vascular smooth muscle cells. Biochem Biophys Res Commun. 1996; 224: 808-14
|
|
|
12) Papadaki M, Tilton RG, Eskin SG, et al. Nitric oxide production by smooth muscle cells: modulation by fluid flow. Am J Physiol. 1998; 273: H6616-26
|
|
|
13) Grabowski EF, Reininger AJ, Petteruti PG, et al. Shear stress decreases endothelial cell tissue factor activity by augmenting secretion of tissue factor pathway inhibitor. Arterioscler Thromb Vasc Biol. 2001; 21: 157-62
|
|
|
14) Papadaki M, Ruef J, Nguyen KT, et al. Differential regulation of protease activated receptor-1 and tissue plasminogen activator expression by shear stress in vascular smooth muscle cells. Circ Res. 1998; 83: 1027-34
|
|
|
15) Malek AM, Jackman R, Rosenberg RD, et al. Endothelial expression of thrombomodulin is reversibly regulated by fluid shear stress. Circ Res. 1994; 74: 852-60
|
|
|
16) Ueba H, Kawakami M, Yaginuma T. Shear stress as an inhibitor of vascular smooth muscle cell proliferation: Role of transforming growth factor- β1 and tissue-type plasminogen activator. Arterioscler Thromb Vasc Biol. 1997; 17: 1512-6
|
|
|
17) Joye JD, Schulman DS, Lasorda D, et al. Intracoronary Doppler guide wire versus stress single-photon emission computed tomographic thallium-201 imaging in assessment of intermediate coronary stenosis. J Am Coll Cardiol. 1994; 24: 940-7
|
|
|
18) Pijls NHJ, De Bruyne B, Peels K, et al. Measurement of myocardial fractional flow reserve to assess the functional severity of coronary artery stenosis. N Engl J Med. 1996; 334: 1703-8
|
|
|
19) Claeys MJ, Vrints CJ, Bosmans HJ, et al. Coronary flow reserve during coronary angioplasty inpatients with a recent myocardial infarction: relation to stenosis and myocardial viability. J Am Coll Cardiol. 1996; 28: 1712-8
|
|
|
20) Bech GJ, De Bruyne B, Bonnier HJ, et al. Long-term follow-up after deferral of percutaneous transluminal coronary angioplasty of interme-diate stenosis on the basis of coronary pressure measurement. J Am Coll Cardiol. 1998; 31: 841- 7
|
|
|