|
1) Backs J, Olson EN. Control of cardiac growth by histone acetylation/deacetylation. Circ Res. 2006; 98: 15-24
|
|
|
|
2) McKinsey TA, Zhang CL, Olson EN. Control of muscle development by dueling HATs and HDACs. Curr Opin Genet Dev. 2001; 11: 497- 504
|
|
|
|
|
3) Lickert H, Takeuchi JK, Von Both I, et al. Baf60c is essential for function of BAF chromatin remodelling complexes in heart development. Nature. 2004; 432: 107-12
|
|
|
|
|
|
4) Miyamoto S, Suzuki T, Muto S, et al. Positive and negative regulation of the cardiovascular transcription factor KLF5 by p300 and the oncogenic regulator SET through interaction and acetylation on the DNA-binding domain. Mol Cell Biol. 2003; 23: 8528-41
|
|
|
|
|
|
5) Suzuki T, Muto S, Miyamoto S, et al. Functional interaction of the DNA-binding transcription factor Sp1 through its DNA-binding domain with the histone chaperone TAF-I. J Biol Chem. 2003; 278: 28758-64
|
|
|
|
|
|
6) Matsumura T, Suzuki T, Aizawa K, et al. The deacetylase HDAC1 negatively regulates the cardiovascular transcription factor Kruppel-like factor 5 through direct interaction. J Biol Chem. 2005; 280: 12123-9
|
|
|
|
|
|
7) Suzuki T, Nishi T, Nagino T, et al. Functional interaction between the transcription factor Kruppel-like factor 5 and poly(ADP-ribose) polymerase-1 in cardiovascular apoptosis. J Biol Chem. 2007; 282: 9895-901
|
|
|
|
|
|
8) Gorgoulis VG, Vassiliou LV, Karakaidos P, et al. Activation of the DNA damage checkpoint and genomic instability in human precancerous lesions. Nature. 2005; 434: 907-13
|
|
|
|
|
|
9) Bartkova J, Horejsi Z, Koed K, et al. DNA damage response as a candidate anti-cancer barrier in early human tumorigenesis. Nature. 2005; 434: 864-70
|
|
|
|
|